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Old 10-20-2018, 12:04 AM #471
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So the C. sativa L. at the conclusion. Is the L Linneus? Lamarck?

Great read!
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Old 10-20-2018, 12:48 AM #472
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Linnaeus
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Old 11-06-2018, 06:42 PM #473
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Newest preprint paper from George Weiblen's UM group working together with Sunrise Genetics. This is fascinating and has profound implications for breeders.

Christopher J Grassa, Jonathan P Wenger, Clemon Dabney, Shane G Poplawski, S Timothy Motley, Todd P Michael, C J Schwartz, George D Weiblen. "A complete cannabis chromosome assembly and adaptive admixture for elevated cannabidiol (CBD) content.

https://doi.org/10.1101/458083

Full paper available here:
https://www.biorxiv.org/content/earl....full.pdf+html
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Old 12-01-2018, 06:15 PM #474
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A physical and genetic map of Cannabis sativa identifies extensive rearrangement at the 1 THC/CBD acid synthase locus

Kaitlin U. Laverty, Jake M. Stout, Mitchell J. Sullivan, Hardik Shah, Navdeep Gill, Larry Holbrook, Gintaras Deikus Robert Sebra Timothy R. HughesJonathan E. Page, and Harm van Bakel

https://genome.cshlp.org/content/ear....full.pdf+html
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Old 12-01-2018, 06:21 PM #475
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https://www.marijuanaventure.com/dec...abis-genetics/

More similar work but their paper is not completed or published.

Last edited by Sam_Skunkman; 12-02-2018 at 05:43 AM..
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Old 12-03-2018, 02:26 AM #476
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"Jonathan E. Page and Jake M. Stout have filed Patent WO2015196275 on the nucleotide sequence encoding the enzyme CBCAS based reagents, and methods for producing cannabinoids and/or altering cannabinoid production."

Lot of distractions/loud room mates at the house now- I can't work this sentence out. Is this a patent on CBCAS or the reagents and method used to get these results....or all resultant reagents that the CBCAS yielded?

Interesting results nonetheless.

Edit: This says it better under Claims

https://patents.google.com/patent/WO2015196275A1/en
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Old 01-07-2019, 10:51 AM #477
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https://www.ncbi.nlm.nih.gov/pubmed/30610611

"Adenosine A2A-Cannabinoid CB1 Receptor Heteromers in the Hippocampus: Cannabidiol Blunts Δ9-Tetrahydrocannabinol-Induced Cognitive Impairment."

Abstract
At present, clinical interest in the plant-derived cannabinoid compound cannabidiol (CBD) is rising exponentially, since it displays multiple therapeutic properties. In addition, CBD can counteract the undesirable effects of the psychoactive cannabinoid Δ9-tetrahydrocannabinol (Δ9-THC) that hinder clinical development of cannabis-based therapies. Despite this attention, the mechanisms of CBD action and its interaction with Δ9-THC are still not completely elucidated. Here, by combining in vivo and complementary molecular techniques, we demonstrate for the first time that CBD blunts the Δ9-THC-induced cognitive impairment in an adenosine A2A receptor (A2AR)-dependent manner. Furthermore, we reveal the existence of A2AR and cannabinoid CB1 receptor (CB1R) heteromers at the presynaptic level in CA1 neurons in the hippocampus. Interestingly, our findings support a brain region-dependent A2AR-CB1R functional interplay; indeed, CBD was not capable of modifying motor functions presumably regulated by striatal A2AR/CB1R complexes, nor anxiety responses related to other brain regions. Overall, these data provide new evidence regarding the mechanisms of action of CBD and the nature of A2AR-CB1R interactions in the brain.
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