Topically I'd suggest dilute Tea tree oil for disinfection. Some mild burning may be associated with the application. It assist with the pliability of the tissue also. In addendum, you may want to find a topical homeopathic application of "Calendula". Use sparingly. Echinacea and other immune system modulators would be useful also. Not sure any would be a silver bullet, but sometimes these disorders are self limiting and will go away on their own spontaneously. The main thing to do is keep down the inflammation and avoid scar tissue development, which can require more sophisticated treatment down the road. You may want to get a broad spectrum Omega 3, 6 & 9 supplement and use it in pharmacologic doses. That will tend to drive down inflammation pan-systemically.
Of additional interest it seems there may be forms of phototherapy viable for this disorder....I suggest you investigate that more fully in your local area with a dermatologist:
UVA1 phototherapy for genital lichen sclerosus.
Beattie PE, Dawe RS, Ferguson J, Ibbotson SH.
Photobiology Unit, Dermatology Department, University of Dundee, Ninewells Hospital and Medical School, Dundee, UK. email@example.com t.nhs.uk
BACKGROUND: Lichen sclerosus (LS) is characterized histologically by an inflammatory T-cell infiltrate, sclerosis and thickening of the dermis, and epidermal atrophy. Ultraviolet (UV) A1 therapy has been shown to be effective in the management of morphea and scleroderma, diseases that have some histological and clinical similarities with LS, and more recently in extragenital LS. AIM: To determine the effectiveness of UVA1 therapy for genital LS. METHODS: Seven women with severe genital LS uncontrolled by ultrapotent topical corticosteroids, with a median age of 62 years (range 48-78) and disease duration of 6-47 years, were treated with UVA1 therapy from a high output source. After completion of UVA1 therapy, a clinician and the patient graded the overall response of symptoms and physical signs. RESULTS: Five patients improved with therapy. Three obtained moderate improvement in overall disease severity and two had minimal improvement. Of these five, one relapsed within 3 months and another after a year. Both had a further course of UVA1 therapy, resulting in minimal improvement in one and moderate improvement in the other. In the remaining three, disease severity had improved to a point where intermittent use of topical corticosteroids resulted in acceptable control. DISCUSSION: UVA1 therapy may be of benefit in the management of vulval LS, a disease that is often poorly responsive to standard therapies. The therapy is well tolerated and could provide an acceptable therapeutic option for patients with severe disease.
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